Neuropathy, also known as peripheral neuropathy, refers to damage or dysfunction of the peripheral nerves—the network of nerves outside the brain and spinal cord. These nerves are responsible for transmitting signals between the central nervous system and the rest of the body, controlling sensations, movements, and organ function. There are numerous causes of neuropathy, including diabetes, autoimmune diseases, infections (such as Lyme disease or HIV), traumatic injuries, and exposure to toxins or certain medications. Depending on the underlying cause and the specific nerves affected, neuropathy can manifest with a wide range of symptoms, including numbness, tingling, weakness, and pain in the affected areas. In severe cases, neuropathy can lead to muscle atrophy, loss of coordination, and impaired organ function. Management of neuropathy focuses on treating the underlying cause, when possible, and addressing symptoms to improve quality of life. This may involve medications to relieve pain and reduce inflammation, lifestyle modifications such as regular exercise and a balanced diet to manage underlying conditions like diabetes, and physical therapy to improve strength, balance, and mobility.
Title : Perception and individuality in patient cases identifying the ongoing evolution of Myalgic Encephalomyelitis/Chronic Fatigue Syndrome (ME/CFS)
Ken Ware, NeuroPhysics Therapy Institute, Australia
Title : Narrative medicine: A communication therapy for the communication disorder of Functional Seizures (FS) [also known as Psychogenic Non-Epileptic Seizures (PNES)]
Robert B Slocum, University of Kentucky HealthCare, United States
Title : Personalized and Precision Medicine (PPM), as a unique healthcare model through biodesign-driven biotech and biopharma, translational applications, and neurology-related biomarketing to secure human healthcare and biosafety
Sergey Victorovich Suchkov, N. D. Zelinskii Institute for Organic Chemistry of the Russian Academy of Sciences, Russian Federation
Title : Neuro sensorium
Luiz Moutinho, University of Suffolk, United Kingdom
Title : GBF1 inhibition reduces amyloid-beta levels in viable human postmortem Alzheimer's disease cortical explant and cortical organoid models
Sean J Miller, Yale School of Medicine, United States
Title : Traumatic Spinal Cord Injuries (tSCI) - Are the radiologically based “advances” in the management of the injured spine evidence-based?
W S El Masri, Keele University, United Kingdom